Mots-clés
> MYOBASE > PRODUITS CHIMIQUES ET BIOLOGIQUES > protéine > récepteur > récepteur membranaire > récepteur synaptique > récepteur cholinergique
récepteur cholinergiqueSynonyme(s)récepteur à l'acétylcholine ;cholinergic receptor ;R-ACh ;ACh-R AChR |
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Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Krejci E ; Akaaboune M | 2005Communication n° 342 At the mammalian neuromuscular junction, Acetylcholinesterase (AChE) controls the temporal stimulation of the muscle by the nerve. AChE is concentrated in the basal lamina and classical experiments suggest that this enzyme i[...]Article
Chevessier F, Auteur ; Faraut B ; Ravel Chapuis A ; Richard P ; Gaudon K ; Bauche S ; Prioleau C ; Herbst R ; Goillot E ; Ioos C ; Azulay JP ; Attarian S ; Leroy JP ; Fournier E ; Legay C ; Schaeffer L ; Koenig J ; Fardeau M ; Eymard B ; Pouget J ; Hantaï D | 2005Article
Shiraishi H ; Motomura M ; Yoshimura T ; Fukudome T ; Fukuda T ; Nakao Y ; Tsujihata M ; Vincent A ; Eguchi K | 2005Accès au résumé PubMed / to PubMed abstractArticle
Losen M ; Stassen MHW ; Martinez-Martinez P ; Machiels BM ; Duimel H ; Frederik PM ; Veldman H ; Wokke JHJ ; Spaans F ; Vincent A | 2005Accès au résumé PubMed / to PubMed abstractArticle
Silva-Barbosa SD ; Butler-Browne GS ; Mouly V | 2005Accès au résumé PubMed / to PubMed abstractArticle
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Müller J ; Stucka R ; Neudecker S ; Zierz S ; Schmidt C ; Huebner A ; Lochmueller H ; Abicht A | 2005Communication n° 264 Objective : Congenital myasthenic syndromes (CMS) are caused by various genetic defects of presynaptic, synaptic and postsynaptic proteins. We report a postsynaptic CMS caused by two compound heterozygous mutations of the CH[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Poulas K ; Sideris S ; Tsiamalos P ; Kostelidou K ; Stergiou C ; Kordas G ; Tzartos SJ | 2005Communication n° 488 Muscle acetylcholine receptor (AChR), the autoantigen in myasthenia gravis, is difficult to be produced in large amounts as an intact molecule. We have successfully expressed soluble extracellular domains (ECDs) of ?, ?, ? a[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Brenner T ; Hamra-Amitay Y ; Sicsic C ; Berrih-Aknin S | 2005Communication n° 106 Myasthenia gravis is an antibody-mediated, autoimmune neuromuscular disease in which the nicotinic acetylcholine receptor (AChR) is the major autoantigen. The typical neuromuscular junction symptoms can be transiently allevi[...]Article
Yu H, Auteur ; Ishigaki K ; Shimahara T ; Bournaud R ; Le Poupon C ; Prado de Carvalho L ; Corringer PJ ; Fardeau M ; Koenig J ; Eymard B ; Hantaï D | 2005Communication n° 253 : Congenital myasthenic syndromes (CMS) are a group of rare genetic disorders that affect neuromuscular transmission. Most of the CMS are caused by mutations of the nicotinic acetylcholine receptor (nAChR) in skeletal muscl[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Nouioua S ; Sifi Y ; Hecham N ; Ali Pacha L ; Richard P ; Grid D ; Hamri A ; Tazir M | 2005Communication n° 440 Introduction : Mutations in various genes expressed at the neuromuscular junction cause congenital myasthenic syndromes (CMS). Currently their classification is physiopathological, implicating 8 genes. Post-synaptic congenit[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Richard P ; Gaudon K ; Haddad H ; Koenig J ; Bauche S ; Grid D ; Gouider-Khouja N ; Mayer M ; Desnuelle C ; Pouget J ; Hentati F ; Eymard B ; Hantaï D | 2005Communication n° 235. Background: Congenital myasthenic syndromes (CMS) are hereditary disorders characterised by a defect in the neuromuscular transmission. Most cases showed recessive mode of inheritance and molecular analyses have shown that [...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Feferman T ; Cizeron-Clairac G ; Fuchs S ; Souroujon M ; Berrih-Aknin S | 2005Communication n° 448 Myasthenia Gravis (MG) is an autoimmune disease mediated by antibodies directed against the acetylcholine receptor (AChR), which are found in about 85% of patients. Autoimmune experimental MG (EAMG) can be induced in rats by[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Colomer J ; Müller JS ; Nascimiento A ; Vernet A ; Lochmuller H | 2005Communication n° 188 Introduction: The majority of postsynaptic congenital myasthenic syndromes (CMS) result from deficiency of acetylcholine receptors (AChR) at the endplate, due to mutations of the gene encoding the epsilon subunit of the AChR[...]Article
Accès au résumé PubMed / to PubMed abstractArticle
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Frenkian-Cuvelier M ; Boneva N ; Clairac G ; Bidault J ; Berrih-Aknin S | 2005Communication n° 512 The Aquired Autoimmune Myasthenia Gravis (MG) is characterized by 3 immunological entities : seropositive MG (SPMG), seronegative MG (SNMG) and MuSK+. Despite the different immunological profile, all MG patients present the [...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Berrih-Aknin S ; Poëa-Guyon S ; Christadoss P ; Le Panse R ; Guyon T ; Bidault J ; Tzartos SJ | 2005Communication n° 590 Myasthenia gravis (MG) is an autoimmune disease associated with thymic pathologies, including hyperplasia. In this study we investigated the processes that may lead to thymic overexpression of the triggering antigen, the ace[...]Article
Fostieri E ; Tzartos SJ ; Berrih-Aknin S ; Beeson D ; Mamalaki A | 2005Accès au résumé PubMed / to PubMed abstractArticle
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Molenaar PC ; Ter Beek P ; Wintzen AR ; Verschuuren J ; Vincent A | 2005Communication n°369 The objective was to develop a mouse model for studying the pathophysiology of myasthenia gravis patients with auto-antibodies to MuSK, a tyrosine kinase receptor responsible for clustering of acetylcholine receptors (AChRs) [...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Méjat A ; Ramond F ; Ravel Chapuis A ; Huze C ; Caussanel S ; Calhabeu F ; Ancelin K ; Gangloff Y ; Risson V ; Goillot E ; Schaeffer L ; Bassel Duby R ; Khochbin S ; Olson E | 2005Communication n° 92 Electrical activity arising from motor innervation influences skeletal muscle physiology by controlling the expression of many muscle genes, including acetylcholine receptor (AChR) subunits genes. How electrical activity is c[...]Thèse/Mémoire
Nancy P, Auteur | 2005La myasthénia gravis est une maladie auto-immune due à la présence d’autoanticorps contre le récepteur nicotinique de l'acétylcholine (RACh) de la jonction neuromusculaire. Cette maladie touche beaucoup de femmes surtout dans la période féconde [...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Aricha R ; Feferman T ; Souroujon M ; Fuchs S | 2005Communication n° 449 Myasthenia gravis (MG) and experimental autoimmune MG (EAMG) are T-cell dependent antibody-mediated autoimmune disorders, in which the nicotinic acetylcholine receptor (AChR) is the major autoantigen. The anti-AChR response [...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Feferman T ; Maiti P ; Berrih-Aknin S ; Bismuth J ; Bidault J ; Fuchs S | 2005Communication n° 312 Myasthenia gravis (MG) and its animal model, experimental autoimmune MG (EAMG) are autoimmune disorders in which the acetylcholine receptor (AChR) is the major autoantigen. Microarray technology was used to identify new pote[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Molgo J ; Adams D ; Colasante C ; Jaimovich E | 2005Communication n° 573 At the neuromuscular junction (NMJ) not yet well explored is the possible role of neuromuscular activity mediated via an IP3 cascade in perisynaptic Schwann cells, and in the postsynaptic elements of the NMJ. The well docume[...]