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Auteur Salmon A |
Documents disponibles écrits par cet auteur (15)
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Article
Delta-sarcoglycan deficiency induces muscular dystrophy and dilated cardiomyopathy (DCM) through pathological mechanisms that remain elusive. Lack of dystrophin, an other sarcolemmal protein, induces skeletal muscle fibers Ca2+ overload that may[...]Article
Fraysse B ; Nagi SM ; Boher B ; Ragot H ; Laine J ; Salmon A ; Fiszman MY ; Toussaint M ; Fromes Y | 2010Accès au résumé PubMed / to PubMed abstractArticle
Mongue-Din H ; Salmon A ; Fiszman MY ; Fromes Y | 2009Accès au résumé PubMed / to PubMed abstractArticle
Sarcolemmopathies are muscular dystrophies caused by defects of proteins located within or near cell membrane. Underlying Pathological mechanisms remain unclear. Nevertheless, in dystrophin deficient mdx mice it has been shown that skeletal musc[...]Article
Congrès international de myologie 2008 (International Congress of Myology 2008; 26-30 mai 2008; Marseille, France) ; Mongue-Din H ; Liu JM ; Salmon A ; Fiszman MY ; Wdzieczak-Bakala J ; Fromes Y | 2008The chronically failing heart is characterized by alterations in tissue structure, particularly fibrous tissue formation, responsible for the loss of myocardial compliance. Furthermore, rhythm disturbances are commonly observed. The underlying a[...]Article
Congrès international de myologie 2008 (International Congress of Myology 2008; 26-30 mai 2008; Marseille, France) ; Decostre V ; Khouzami L ; Varnous S ; Caramelle P ; Perier M ; Adamy C ; Arimura T ; Salmon A ; Enond C ; Meune C ; Pecker F ; Bonne G | 2008Emery-Dreifuss muscular dystrophy (EDMD) is characterized by cardiac dysfunction and dilation associated with skeletal muscle defects. It is caused by mutations in the LMNA gene encoding the nuclear proteins lamin A/C that form a meshwork under [...]Article
Congrès international de myologie 2008 (International Congress of Myology 2008; 26-30 mai 2008; Marseille, France) ; Roques C ; Bouchemal K ; Salmon A ; Fattal E ; Fromes Y | 2008Gene transfer is an interesting therapeutic approach for inherited muscular dystrophies with no curative treatment currently available. Nevertheless, DNA does not freely cross the membranes as it is a hydrophilic, negatively charge macromolecule[...]Article
Roques C ; Salmon A ; Fiszman MY ; Fattal E ; Fromes Y | 2007Accès au résumé PubMed / to PubMed abstractArticle
Mongue-Din H ; Salmon A ; Fiszman MY ; Fromes Y | 2007Accès au résumé PubMed / to PubMed abstractArticle
Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Serose A ; Prudhon B ; Salmon A ; Doyennette-Moyne MA ; Fiszman M ; Fromes Y ; Doyennette MA | 2005Communication n° 202 Introduction : As cardiac disorders are a common complication in neuromuscular disorders, it seems essential to treat both skeletal and cardiac muscles in these pathologies. Dilated cardiomyopathies (DCM) are due to progress[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Mongue-Din H ; Salmon A ; Fiszman M ; Fromes Y | 2005Communication n° 201 Familial myopathies are due to primary molecular alterations of striated skeletal and cardiac cells. During cardiomyopathies evolution, patients develop secondary alterations characterized by an abnormal cardiac electric act[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Roques C ; Salmon A ; Serose A ; Fiszman M ; Fattal E ; Fromes Y | 2005Communication n° 203 Background : Concerning myopathies, the treatment of cardiac impairment raises specific issues due to the structure and function of the myocardium. Thus, original routes of administration can be considered as direct injectio[...]Article
Serose A ; Prudhon B ; Salmon A ; Doyennette-Moyne MA ; Fiszman MY ; Fromes Y | 2004Accès au résumé Pubmed/to pubmed abstractArticle
Blair E ; Redwood C ; Oliveira M ; Broxholme J ; Kerr B ; Salmon A ; Ostman-Smith I ; Watkins H ; Ashrafian H | 2001