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Auteur Askanas V

Documents disponibles écrits par cet auteur (40)

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Article

Biochimica et biophysica acta, 1852, 4. Sporadic inclusion-body myositis: A degenerative muscle disease associated with aging, impaired muscle protein homeostasis and abnormal mitophagy

Askanas V ; Engel WK ; Nogalska A | 2015

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En ligne

Article

Journal of neuropathology and experimental neurology, 74, 6. Activation of the Unfolded Protein Response in Sporadic Inclusion-Body Myositis but Not in Hereditary GNE Inclusion-Body Myopathy

Nogalska A, Auteur ; D'Agostino C ; Engel WK ; Cacciottolo M ; Asada S ; Mori K ; Askanas V | 2015

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En ligne

Article

Neurobiology of disease, 65. Sodium phenylbutyrate reverses lysosomal dysfunction and decreases amyloid-β42 in an in vitro-model of inclusion-body myositis

Nogalska A ; D'Agostino C ; Engel WK ; Askanas V | United States | 05/2014

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En ligne

Article

Neuropathology and applied neurobiology, 39, 7. Chaperone-mediated autophagy components are upregulated in sporadic inclusion-body myositis muscle fibres

Cacciottolo M ; Nogalska A ; D'Agostino C ; Engel WK ; Askanas V | 2013

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Livre

Muscle aging, inclusion-body myositis and myopathies

Askanas V ; Engel K | Oxford : Wiley Blackwell | 02/2012

La faiblesse musculaire est inévitable lors du vieillissement, dès 40 ans. Elle peut provoquer une diminution de la mobilité, une augmentation du risque de chute et même le décès. Mais "vous vieillissez" n'est pas un diagnostic suffisant. Les ca[...]

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Document numérique

Article

Acta neuropathologica, 114, 5. NOGO is increased and binds to BACE1 in sporadic inclusion-body myositis and in AbetaPP-overexpressing cultured human muscle fibers

Wojcik S ; Engel WK ; Yan R ; McFerrin J ; Askanas V | 2007

Accès au résumé PubMed / to PubMed abstract

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Article

Current opinion in rheumatology, 19, 6. Inclusion-body myositis, a multifactorial muscle disease associated with aging : current concepts of pathogenesis

Askanas V ; Engel WK | 2007

Accès au résumé PubMed / to PubMed abstract

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Article

Experimental neurology, 240. Endoplasmic reticulum stress induces myostatin precursor protein and NF-kappaB in cultured human muscle fibers : relevance to inclusion body myositis

Nogalska A ; Wojcik S ; Engel WK ; McFerrin J ; Askanas V | 2007

Accès au résumé PubMed / to PubMed abstract

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Article

Neurology Parkin might play a novel role in the pathogenesis of sporadic inclusion-body myositis (s-IBM) (Abstract in 58th Annual Meeting of American Academy of Neurology)

58th Annual Meeting of American Academy of Neurology (April 1-8, 2006; San Diego (California)) ; Paciello O ; Wojcik S ; Engel WK ; Askanas V | 03/2006

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Article

Neurology Proteasome inhibition and endoplasmic reticulum (ER) stress influence aggregation and expression of valosin-containing peptide (VCP) in an inclusion-body-myopathy human-muscle tissue-culture (IBM-HM-TC) model (Abstract in 58th Annual Meeting of American Academy of Neurology)

58th Annual Meeting of American Academy of Neurology (April 1-8, 2006; San Diego (California)) ; McFerrin J ; Engel WK ; Askanas V | 03/2006

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Article

Microscopy research and technique, 67, 3-4. Molecular pathology and pathogenesis of inclusion-body myositis

Askanas V ; Engel WK | 2005

Accès au résumé PubMed / to PubMed abstract

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Article

Neurology Amyloid-beta42 is preferentially deposited in muscle biopsies of patients with sporadic inclusion-body myositis (s-IBM)

55th Annual Meeting program (American Academy of Neurology; March 29 - April 5, 2003; Honolulu, Hawaii) ; Vattemi G ; Checler F ; Engel WK ; Askanas V | 03/2003

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Article

Neurology The Val122IIe mutation in the transthyretin gene predisposes to accumulation of amyloid-beta (ABeta) and development of the inclusion-body myositis (IBM) phenotype in cultured human muscle fibers

55th Annual Meeting program (American Academy of Neurology; March 29 - April 5, 2003; Honolulu, Hawaii) ; McFerrin J ; Engel WK ; Vattemi G ; Askanas V | 03/2003

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Article

Current opinion in rheumatology, 15, 6. Proposed pathogenetic cascade of inclusion-body myositis : importance of amyloid-beta, misfolded proteins, predisposing genes, and aging

Askanas V ; Engel WK | 2003

Accès au résumé PubMed / to PubMed abstract

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Article

Neurology, 61, 2. Transthyretin Val122Ile, accumulated A-beta, and inclusion-body myositis aspects in cultured muscle

Askanas V ; Engel WK ; McFerrin J ; Vattemi G | 2003

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Article

Acta myologica, 21. Inclusion-body myositis : pathogenic role of amyloid-Beta, cholesterol, relation to aging and Alzheimer disease

Askanas V ; Engel WK | 12/2002

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Article

Acta myologica, 21. Newest pathogenic consideration in inclusion-body myositis : Importance of cholesterol and related mechanisms

Sixth Congress of the Mediterranean Society of Myology (May 23-26 2002; Corfu, Greece) ; Askanas V ; Engel WK | 04/2002

Abstract

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Article

Current opinion in neurology, 15. Inclusion-body myositis and myopathies : different etiologies, possibly similar pathogenic mechanisms

Askanas V ; Engel WK | 2002

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Article

Current rheumatology reports, 4, 5. Newest pathogenetic considerations in inclusion-body myositis : Possible role of Amyloid-Beta, cholesterol, relation to aging and to Alzheimer's disease

Askanas V ; Engel K | 2002

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Article

Neurology, 58, 3. Three lipoprotein receptors and cholesterol in inclusion-body myositis muscle

Jaworska-Wilczynska M ; Wilczynski GM ; Engel WK ; Strickland DK ; Weisgraber KH ; Askanas V | 2002

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Article

NeuroReport, 12, 9. Novel cytoplasmic immunolocalization of RNA polymerase II in inclusion-body myositis muscle

Wilczynski GM ; Engel WK ; Askanas V | 07/2001

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Article

Neurology Novel proposed role of glycogen synthase kinase-3Beta (GSK-3Beta) in the pathogenesis of inclusion-body myositis (IBM)

Wilczynski GM ; Broccolini A ; Engel WK ; Askanas V | 04/2001

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Article

Archives of neurology, 55. Sporadic inclusion-body myositis and hereditary Inclusion-body myopathies. Diseases of oxidative stress and aging ?

Askanas V ; Engel KW | 07/1998

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Article

American journal of pathology (The), 152, 4. Light and electron microscopic immunolocalization of presenilin 1 in abnormal muscle fibers of patients with sporadic inclusion-body myositis and autosomal recessive inclusion body myopathy

Askanas V ; Engel WK ; Yang CC ; Alvarez RB ; Lee VMY ; Wisniewski T | 1998

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Article

Brain, 121, 6. Nitric oxide-induced oxidative stress in autosomal recessive and dominant inclusion-body myopathies

Yang CC ; Alvarez RB ; Engel WK ; Heller SL ; Askanas V | 1998

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Auteur Askanas V

Documents disponibles écrits par cet auteur (40)

Biochimica et biophysica acta, 1852, 4. Sporadic inclusion-body myositis: A degenerative muscle disease associated with aging, impaired muscle protein homeostasis and abnormal mitophagy

Journal of neuropathology and experimental neurology, 74, 6. Activation of the Unfolded Protein Response in Sporadic Inclusion-Body Myositis but Not in Hereditary GNE Inclusion-Body Myopathy

Neurobiology of disease, 65. Sodium phenylbutyrate reverses lysosomal dysfunction and decreases amyloid-β42 in an in vitro-model of inclusion-body myositis

Neuropathology and applied neurobiology, 39, 7. Chaperone-mediated autophagy components are upregulated in sporadic inclusion-body myositis muscle fibres

Muscle aging, inclusion-body myositis and myopathies

Acta neuropathologica, 114, 5. NOGO is increased and binds to BACE1 in sporadic inclusion-body myositis and in AbetaPP-overexpressing cultured human muscle fibers

Current opinion in rheumatology, 19, 6. Inclusion-body myositis, a multifactorial muscle disease associated with aging : current concepts of pathogenesis

Experimental neurology, 240. Endoplasmic reticulum stress induces myostatin precursor protein and NF-kappaB in cultured human muscle fibers : relevance to inclusion body myositis

Neurology Parkin might play a novel role in the pathogenesis of sporadic inclusion-body myositis (s-IBM) (Abstract in 58th Annual Meeting of American Academy of Neurology)

Neurology Proteasome inhibition and endoplasmic reticulum (ER) stress influence aggregation and expression of valosin-containing peptide (VCP) in an inclusion-body-myopathy human-muscle tissue-culture (IBM-HM-TC) model (Abstract in 58th Annual Meeting of American Academy of Neurology)

Microscopy research and technique, 67, 3-4. Molecular pathology and pathogenesis of inclusion-body myositis

Neurology Amyloid-beta42 is preferentially deposited in muscle biopsies of patients with sporadic inclusion-body myositis (s-IBM)

Neurology The Val122IIe mutation in the transthyretin gene predisposes to accumulation of amyloid-beta (ABeta) and development of the inclusion-body myositis (IBM) phenotype in cultured human muscle fibers

Current opinion in rheumatology, 15, 6. Proposed pathogenetic cascade of inclusion-body myositis : importance of amyloid-beta, misfolded proteins, predisposing genes, and aging

Neurology, 61, 2. Transthyretin Val122Ile, accumulated A-beta, and inclusion-body myositis aspects in cultured muscle

Acta myologica, 21. Inclusion-body myositis : pathogenic role of amyloid-Beta, cholesterol, relation to aging and Alzheimer disease

Acta myologica, 21. Newest pathogenic consideration in inclusion-body myositis : Importance of cholesterol and related mechanisms

Current opinion in neurology, 15. Inclusion-body myositis and myopathies : different etiologies, possibly similar pathogenic mechanisms

Current rheumatology reports, 4, 5. Newest pathogenetic considerations in inclusion-body myositis : Possible role of Amyloid-Beta, cholesterol, relation to aging and to Alzheimer's disease

Neurology, 58, 3. Three lipoprotein receptors and cholesterol in inclusion-body myositis muscle

NeuroReport, 12, 9. Novel cytoplasmic immunolocalization of RNA polymerase II in inclusion-body myositis muscle

Neurology Novel proposed role of glycogen synthase kinase-3Beta (GSK-3Beta) in the pathogenesis of inclusion-body myositis (IBM)

Archives of neurology, 55. Sporadic inclusion-body myositis and hereditary Inclusion-body myopathies. Diseases of oxidative stress and aging ?

American journal of pathology (The), 152, 4. Light and electron microscopic immunolocalization of presenilin 1 in abnormal muscle fibers of patients with sporadic inclusion-body myositis and autosomal recessive inclusion body myopathy

Brain, 121, 6. Nitric oxide-induced oxidative stress in autosomal recessive and dominant inclusion-body myopathies