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Safety and efficacy of intravenous bimagrumab in inclusion body myositis (RESILIENT): a randomised, double-blind, placebo-controlled phase 2b trial
Hanna MG, Badrising UA, Benveniste O, et al.
The Lancet. Neurology, 2019, 18, 9, p 834
Focused HLA analysis in Caucasians with myositis identifies significant associations with autoantibody subgroups.
Rothwell S, Chinoy H, Lamb JA, et al.
Annals of the rheumatic diseases, 2019, 78, n7, p 996
Community exercise is feasible for neuromuscular diseases and can improve aerobic capacity
Wallace A, Wallace A, Pietrusz A, et al.
Neurology, 2019, 92, 15
Update on outcome assessment in myositis
Rider LG, Aggarwal R, Machado PM, et al.
Nature reviews. Rheumatology, 2018, 14, 5, p 303
Cytosolic 5'-nucleotidase 1A autoantibody profile and clinical characteristics in inclusion body myositis
Lilleker JB, Rietveld A, Pye SR, et al.
Annals of the rheumatic diseases, 2017, 76, 5, p 862
Immune-array Analysis in Sporadic Inclusion Body Myositis Reveals HLA-DRB1 Amino Acid Heterogeneity across the Myositis Spectrum
Rothwell S, Cooper RG, Lundberg IE, et al.
Arthritis & rheumatology (Hoboken, N.J.), 2017, 69, 5, p 1090
Downregulation of myostatin pathway in neuromuscular diseases may explain challenges of anti-myostatin therapeutic approaches
Mariot V, Joubert R, Hourde C, et al.
Nature communications, 2017, 8, 1
Dense genotyping of immune-related loci in idiopathic inflammatory myopathies confirms HLA alleles as the strongest genetic risk factor and suggests different genetic background for major clinical subgroups
Rothwell S, Cooper RG, Lundberg IE, et al.
Annals of the rheumatic diseases, 2016, 75, 8, p 1558
Rare variants in SQSTM1 and VCP genes and risk of sporadic inclusion body myositis
Gang Q, Bettencourt C, Machado PM, et al.
Neurobiology of aging, 2016, 47, p 218.e1
Targeting protein homeostasis in sporadic inclusion body myositis
Ahmed M, Machado PM, Miller A, et al.
Science translational medicine, 2016, 8, 331, p. 1-12
Dense genotyping of immune-related loci in idiopathic inflammatory myopathies confirms HLA alleles as the strongest genetic risk factor and suggests different genetic background for major clinical subgroups.
Rothwell S, Cooper RG, Lundberg IE, et al.
Annals of the rheumatic diseases, 2015, Epub, p. 1-9
Sporadic inclusion body myositis: new insights and potential therapy
Machado PM, Dimachkie MM, Barohn RJ
Current opinion in neurology, 2014, 27, 5, p 591
Ongoing developments in sporadic inclusion body myositis
Machado PM, Ahmed M, Brady S, et al.
Current rheumatology reports, 2014, 16, 12
