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Auteur Parolini D |
Documents disponibles écrits par cet auteur (15)
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Sitzia C, Auteur ; Farini A ; Jardim L ; Razini P ; Belicchi M ; Cassinelli L ; Villa C ; Erratico S ; Parolini D ; Bella P ; da Silva Bizario JC ; Garcia L ; Dias-Baruffi M ; Meregalli M ; Torrente Y | 2016Article
Meregalli M ; Navarro C ; Sitzia C ; Farini A ; Montani E ; Wein N ; Razini P ; Beley C ; Cassinelli L ; Parolini D ; Belicchi M ; Parazzoli D ; Garcia L ; Torrente Y | 2013Article
4th International Congress of Myology, 4ème colloque international de Myologie (9-13 mai 2011; Lille (France)) ; Sitzia C ; Farini A ; Navarro C ; D'Antona G ; Belicchi M ; Parolini D ; Bottinelli R ; Meregalli M ; Torrente Y | AFM-TELETHON | 2011Limb Girdle Muscular Dystrophies (LGMDs) are a group of muscular diseases characterized by predominant weakness and wasting of muscles of the pelvic and shoulder girdle. LGMD-2B and MM were found to arise from defects in the dysferlin gene. In L[...]Article
Erratico S ; Villa C ; Razini P ; Cassinelli LM ; Parolini D ; Meregalli M ; Belicchi M ; Torrente Y | AFM-TELETHON | 2011Peripheral blood is a promising alternative source of stem cells for transplantation for the treatment of different malignancies. Using CD133, as a marker of stemness, we identified a subpopulation of purified blood-derived stem cells which diff[...]Article
4th International Congress of Myology, 4ème colloque international de Myologie (9-13 mai 2011; Lille (France)) ; Del Fraro G ; Meregalli M ; Farini A ; Belicchi M ; Parolini D ; Razini P ; Cassinelli L ; Angeloni V ; Maciotta S ; Bresolin N ; de Silva Bizario J ; Garcia L ; Torrente Y | 2011In DMD, skeletal and cardiac muscles are affected, leading to wheelchair dependency, respiratory failure and premature death. A combination of different strategies might enhance the possibility of successful therapy. We isolated CD133+ cells fro[...]Article
Farini A ; Belicchi M ; Parolini D ; Sitzia C ; Cassinelli L ; Del Fraro G ; Razini P ; Angeloni V ; Jardim L ; da Silva Bizario J ; Garcia L ; Torrente I | AFM-TELETHON | 2011In DMD, skeletal and cardiac muscles are affected, leading to wheelchair dependency, respiratory failure and premature death. Recent advances have pointed out a variety of possible therapeutic approaches. A combination of these strategies might [...]Article
Meregalli M ; Farini A ; Parolini D ; Maciotta S ; Torrente Y | 2010Accès au résumé PubMed / to PubMed abstractArticle
Marchesi C ; Belicchi M ; Meregalli M ; Farini A ; Cattaneo A ; Parolini D ; Gavina M ; Porretti L ; D'Angelo MG ; Bresolin N ; Torrente Y | 05/2008Article
Belicchi M ; Meregalli M ; Marchesi C ; Lopa R ; Porretti L ; Parolini D ; D'Angelo MG ; Bresolin N ; Torrente Y | 2008Although the natural history of patients with Duchenne Muscular dystrophy (DMD) is characterized by a progressive impairment of muscle function leading to death for cardio-pulmonary failure, there is a clinical variability in these patients rega[...]Article
Meregalli M ; Farini A ; D'Antona G ; Belicchi M ; Goyenvalle A ; Parolini D ; Bresolin N ; Bottinelli R ; Garcia L ; Torrente Y | 2008Several recent advances have pointed out a variety of possible therapeutic approaches in muscular dystrophies, from pharmacological treatments to gene therapy and cell therapy with different types of newly identified stem cells. A combination of[...]Article
Congrès international de myologie 2008 (International Congress of Myology 2008; 26-30 mai 2008; Marseille, France) ; Navarro C ; Farini A ; Meregalli M ; Belicchi M ; Parolini D ; Razini P ; Krahn M ; Wein N ; Bourg N ; Bartoli M ; Richard I ; Torrente Y | 2008Mutations in gene encoding Dysferlin are involved in Limb-gird Muscular Dystrophy type 2B (LGMD-2B) and and Miyoshi myopathy (MM), both diseases are characterized by progressive weakness and wasting of skeletal muscles. Dysferlin is abundantly e[...]Article
Congrès international de myologie 2008 (International Congress of Myology 2008; 26-30 mai 2008; Marseille, France) ; Maciotta S ; Meregalli M ; Farini A ; Belicchi M ; Parolini D ; Bresolin N ; Torrente Y | 2008DMD is caused by frameshift mutations in the gene encoding for dystrophin. These mutations are responsible for the loss of function of the dystrophin protein that leads to membrane destabilization and subsequent activation of pathophysiological [...]Article
Congrès international de myologie 2008 (International Congress of Myology 2008; 26-30 mai 2008; Marseille, France) ; Parolini D ; Meregalli M ; Belicchi M ; Farini A ; Razini P ; Maciotta S ; Torrente Y | 2008Duchenne muscular dystrophy (DMD) is a progressive neuromuscular disease due to a deficiency in dystrophin, a 427kDa protein located at the sarcolemma and acting as a linker between cytoskeleton and extracellular matrix Several observations sugg[...]Article
Farini A ; Meregalli M ; Belicchi M ; Battistelli M ; Parolini D ; D'Antona G ; Gavina M ; Ottoboni L ; Constantin G ; Bottinelli R ; Torrente Y | 2007Accès au résumé PubMed / to PubMed abstract 21/09/2007 - Rôle clef des lymphocytes dans le développement de la fibrose chez la souris mdx La dystrophie musculaire de Duchenne (DMD) est une maladie génétique due à un déficit en dyst[...]Article
Gavina M ; Belicchi M ; Rossi B ; Ottoboni L ; Colombo F ; Meregalli M ; Battistelli M ; Forzenigo L ; Biondetti P ; Pisati F ; Parolini D ; Farini A ; Issekutz AC ; Bresolin N ; Rustichelli F ; Constantin G ; Torrente Y | 15/10/2006Accès au résumé PubMed / to PubMed abstract