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Auteur Clairac G |
Documents disponibles écrits par cet auteur (5)
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Article
Maurer M, Auteur ; Bougoin S ; Feferman T ; Frenkian M ; Bismuth J ; Mouly V ; Clairac G ; Tzartos S ; Fadel E ; Eymard B ; Fuchs S ; Souroujon MC ; Berrih-Aknin S | 2015Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Le Panse R ; Clairac G ; Truffault F ; Berrih-Aknin S | 2005Communication n° 506 Myasthenia Gravis (MG) is an autoimmune disease due to autoantibodies directed against components of the neuromuscular junction and leading to disabling fatigability of muscles. Our objective, using microarrays, was to ident[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Frenkian-Cuvelier M ; Boneva N ; Clairac G ; Bidault J ; Berrih-Aknin S | 2005Communication n° 512 The Aquired Autoimmune Myasthenia Gravis (MG) is characterized by 3 immunological entities : seropositive MG (SPMG), seronegative MG (SNMG) and MuSK+. Despite the different immunological profile, all MG patients present the [...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Meraouna A ; Clairac G ; Le Panse R ; Bismuth J ; Berrih-Aknin S | 2005Communication n° 471 Myasthenia Gravis (MG) is caused by autoantibodies against the acetylcholine receptor (AChR) and is often associated with thymic abnormalities. The hyperplastic thymus contains germinal centres (GCs), with B cells producing [...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Clairac G ; Meraouna A ; Le Panse R ; Berrih-Aknin S | 2005Communication n° 507 Myasthenia Gravis (MG) is associated with thymic hyperplasia characterized by germinal center formation in the thymus. The glucocorticoids, widely used in the MG treatment, induce remodeling and cell distribution modificatio[...]