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Auteur Rendon A |
Documents disponibles écrits par cet auteur (33)
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Vacca O ; Zarrouki F ; Izabelle C ; Belmaati Cherkaoui M ; Rendon A ; Dalkara D ; Vaillend C | 20/04/2024![]()
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Barboni MTS ; Joachimsthaler A ; Roux MJ ; Nagy ZZ ; Ventura DF ; Rendon A ; Kremers J ; Vaillend C | England | 17/11/2022![]()
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Barboni MTS ; Liber AMP ; Joachimsthaler A ; Saoudi A ; Goyenvalle A ; Rendon A ; Roger JE ; Ventura DF ; Kremers J ; Vaillend C | United States | 05/2021![]()
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Belmaati Cherkaoui M ; Vacca O ; Izabelle C ; Boulay AC ; Boulogne C ; Gillet C ; Barnier JV ; Rendon A ; Cohen-Salmon M ; Vaillend C | United States | 28/11/2020![]()
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Brignol TN, Auteur ; Fort PE ; Ventura DF ; Tadayoni R ; Rendon A | 26/09/2018To the Editor: We read with interest the recent article by Rice and colleagues1 reporting on 596 patients with Duchenne muscular dystrophy (DMD) treated with steroid and evaluated for cataract development. The authors reported that the prevalenc[...]![]()
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Brignol TN, Auteur ; Fort PE ; Ventura DF ; Tadayoni R ; Rendon A | 2018Comment in: Reply. [J AAPOS. 2018] Comment on: Cataract development associated with long-term glucocorticoid therapy in Duchenne muscular dystrophy patients. [J AAPOS. 2018]![]()
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Aragón J, Auteur ; González-Reyes M ; Romo-Yáñez J ; Vacca O ; Aguilar Gonzalez G ; Rendon A ; Vaillend C ; Montanez C | 2018![]()
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El Mathari B, Auteur ; Sene A ; Charles-Messance H ; Vacca O ; Guillonneau X ; Grepin C ; Sennlaub F ; Sahel JA ; Rendon A ; Tadayoni R | 2015![]()
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Benabdesselam R ; Dorbani-Mamine L ; Benmessaoud-Mesbah O ; Rendon A ; Mhaouty-Kodja S ; Hardin-Pouzet H | 2012Accès au résumé PubMed / to PubMed abstract![]()
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Tadayoni R ; Rendon A ; Soria-Jasso LE ; Cisneros B | 2012Accès au résumé PubMed / to PubMed abstract![]()
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4th International Congress of Myology, 4ème colloque international de Myologie (9-13 mai 2011; Lille (France)) ; Benard R ; Giocanty-Auregan A ; Chaine G ; El-Mathari B ; Sahel JA ; Rendon A ; R.Tadayoni | 2011Purpose: To explore the interactions between Dystrophin protein 71 (Dp71), a sub-membranous cytoskeleton protein, and the blood retinal barrier (BRB), and a possible protective effect of dexamethasone through the regulation of Dp71. Methods: An [...]![]()
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Congrès international de myologie 2008 (International Congress of Myology 2008; 26-30 mai 2008; Marseille, France) ; Benard R ; Dupas B ; Tadayoni R ; Sene A ; Roux MJ ; Nudel U ; Yaffe D ; Sahel JA ; Rendon A | 2008Dp71 is the most abundant Duchenne Muscular Dystrophy (DMD) gene product expressed in the retina. This protein in the Müller glial cells (MGC) plays a role in regulating the retinal homeostasis by clustering Kir4.1 and AQP4 channels. (see poster[...]![]()
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Congrès international de myologie 2008 (International Congress of Myology 2008; 26-30 mai 2008; Marseille, France) ; Sene A ; Fort PE ; Forster V ; Mornet D ; Nudel U ; Yaffe D ; Sahel JA ; Rendon A | 2008We have showed that Dp71 and Utrophin are the only dystrophin superfamily members expressed in Müller Glial cells (MGC). Dp71 deletion leads to a compensating utrophin up-regulation Here we characterized the Dp71 or Utrophin /Dystrophin Associat[...]![]()
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Accès au résumé PubMed / to PubMed abstract![]()
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Fort P ; Estrada FJ ; Bordais A ; Mornet D ; Sahel JA ; Picaud S ; Vargas HR ; Coral-Vazquez RM ; Rendon A | 2005![]()
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Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Cerecedo D ; Martinez D ; Chavez O ; Mornet D ; Rendon A ; Mondragón R | 2005Communication n° 256. Platelet cytoskeleton is constituted by a complex group of proteins responsible for the dramatic changes in cell shape and volume that occur during platelet activation. This process, which involves both granular secretion a[...]![]()
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Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Vaillend C ; Rosenvald Y ; Fort P ; Candelario A ; Mornet D ; Guégan M ; Martinez-Rojas D ; Rendon A ; Nudel U ; Yaffe D ; Laroche S | 2005Communication n° 620 Mutations affecting Dp71, major product of the dystrophin gene in the brain, are thought to be responsible for the most severe cases of mental retardation in DMD. To investigate this hypothesis, we evaluated the biochemical [...]![]()
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Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Hernandez-Gonzalez E ; Martinez-Rojas D ; Mornet D ; Rendon A | 2005Communication n° 220. Introduction : Dystrophin is a member of the protein family coded by the Duchenne muscular dystrophy gene, which is expressed in muscular and non-muscular tissues. The absence of dystrophin alters the dystrophin-associated [...]![]()
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Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Fort P ; Tadayoni R ; Yaffe D ; Nudel U ; Pannicke T ; Reichenbach A ; Sahel JA ; Paques M ; Rendon A | 2005Communication n° 524 Introduction : The dystrophin protein Dp71 is the major Duchenne muscular dystrophy (DMD) gene product in non-muscle tissues. In spite of the fact that Dp71 is required for the organization of dystrophin-associated protein c[...]![]()
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Chavez O, Auteur ; Chavez O ; Harricane MC ; Aleman V ; Dorbani L ; Larroque C ; Mornet D ; Rendon A ; Martinez-Rojas D | 2000![]()
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Vaillend C ; Billard JM ; Claudepierre T ; Rendon A ; Dutar P ; Ungerer A | 1998Accès au résumé PubMed / to PubMed abstract